HIV/AIDS Skepticism

53c5db81627a583e1bbf Pointing to evidence that HIV is not the necessary and sufficient cause of AIDS

Archive for the ‘antiretroviral drugs’ Category

Questioning HIV/AIDS

Posted by Henry Bauer on 2015/02/21

A few years ago I mentioned a research project ” “to examine the factors involved in making the decision not to take HAART or to discontinue its use”, conducted by Dr. Goodson at Texas A&M University.

A few months ago Goodson published an excellent summary of the questions that HIV/AIDS theory has yet to answer: “Questioning the HIV-AIDS hypothesis: 30 years of dissent”. As one would expect, HIV/AIDS vigilantes went into attack mode and urged the publishers to withdraw the article.

To their credit, after weeks of undisclosed consideration, they did not withdraw it, merely changed its category from “Hypothesis and Theory” to “Opinion” — a difference without a distinction, or something like that. They also published a “Commentary” from Seth Kalichman that illustrates his penchant for ad hominem rants lacking substantive argument. You can access that from a link at the right of the Goodson piece. Lots of comments, but some of them have been censored.

In case you never came across it before, do read how Kalichman reminded me of the tale of Dr. Jekyll and Mr. Hyde.


And here’s a long blog post from yet another person who has realized that HIV/AIDS is naked untruth: Nobel Prize Winner In 1994 “HIV Doesn’t Cause AIDS”.




Posted in antiretroviral drugs, HIV does not cause AIDS, HIV skepticism | Tagged: , | 3 Comments »

Manslaughter by PreEposure Prophylaxis

Posted by Henry Bauer on 2014/07/13

The HIV/AIDS Establishment — Big Pharma, NIAID, etc. etc. — is assiduously promulgating the idea that healthy individuals who engage in sex should imbibe highly toxic substances so that they will be less likely to become “HIV-positive”.

This illustrates how true believers and those with vested interests are able to bias clinical trials to deliver desired results even when much earlier data already established that the desired results cannot have been obtained honestly:  for example, several trials of tenofovir to prevent “HIV infection” managed to report that serious adverse events from tenofovir were no more common than from placebo, even as it has long been established that tenofovir causes kidney failure and other harm.

Since this illustrates general flaws in medicine and science, I posted the full analysis on my scimedskeptic blog rather than here; see When prophecy fails.

Posted in antiretroviral drugs, clinical trials, experts, HIV absurdities, HIV risk groups, HIV transmission, sexual transmission, uncritical media | Tagged: , , , | 5 Comments » and its origin

Posted by Henry Bauer on 2014/06/19

When a website labels itself as some sort of “truth”, it’s natural to wonder why its creators feel the need to do so. If it actually conveys truth, that would soon become obvious. In this case, the opposite soon became obvious. was set up by vigilantes determined to defend the indefensible HIV = AIDS dogma, which is perpetually endangered as “lifesaving” drugs disable and kill and as absurdities multiply.

The immediate impetus for establishing had been Celia Farber’s article, “Out of Control: AIDS and the Corruption of Medical Science” (Harper’s, March 2006), which described the death of a pregnant woman, Joyce Hafford, who had been participating in a clinical trial of “antiretroviral” drugs to determine what the “treatment-limiting toxicities” might be. One wonders how any Human Subjects Review Board anywhere would approve such a trial, let alone a trial that enrolled pregnant women. In plainer words, such an experiment administers toxic substances in increasing amounts until signs of toxic harm become obvious.
The drugs being compared were Viracept (nelfinavir) and Viramune (nevirapine), which were used in combination with AZT (Retrovir, zidovudine) and Epivir (lamivudine). The toxicities of all those drugs were well known before Hafford was subjected to them in 2003.
The deadly toxicity of AZT was particularly well known. It was the first “antiretroviral” drug, and some 150,000 “HIV-positive” individuals had died under AZT “treatment” over about a decade (“HAART saves lives — but doesn’t prolong them!?”).
As for “Nevirapine (Viramuneä)”, the NIH Treatment Guidelines (4 February 2002) had already described its life-threatening “side” effects:
“ • Severe, life-threatening hepatotoxicity, including fulminant and cholestatic hepatitis, hepatic necrosis, and hepatic failure. Patients should be advised to seek medical evaluation immediately should signs and symptoms of hepatitis occur.
• Severe, life-threatening, and even fatal skin reactions, including Stevens-Johnson syndrome, toxic epidermal necrolysis, and hypersensitivity reactions characterized by rash, constitutional findings, and organ dysfunction have occurred with nevirapine treatment”.

But vigilante defenders of HIV/AIDS orthodoxy were distraught that such a respectable mainstream magazine as Harper’s would bring to wide attention the truth about the reckless manner in which toxic “antiretroviral drugs” are tested on unwary individuals and subsequently prescribed to equally unwary “HIV-infected” individuals.’s “website was developed in March, 2006, by Bob Funkhouser of Los Alamos National Laboratory, Nathan Geffen of The Treatment Action Campaign, Dr. John P. Moore of Weill Medical College of Cornell University, and Dr. Bette Korber of Los Alamos National Laboratory. It is hosted by The Treatment Action Campaign” (Wayback Machine snapshot, 16 March 2006).

It is worth noting that these and other HIV/AIDS vigilantes habitually denigrate statements from people like Peter Duesberg by pointing out that they have never actually done research on HIV or on AIDS. But among the AIDStruth groupies, only Moore has the distinction of being an actual researcher in this field, and his accomplishments include a completely unsuccessful search for an HIV microbicide and the synthesis of an “HIV” that self-destructed spontaneously (Layne SP, Merges MJ, Dembo M, Spouge JL, Conley SR, Moore JP, Raina JL, Renz H, Gelderblom HR, Nara PL, “Factors underlying spontaneous inactivation and susceptibility to neutralization of human immunodeficiency virus”, Virology, 189 [1992] 695-714).

By 3 January 2007, there had been added to the AIDStruth team the economist Professor Nicoli Nattrass, Director of the AIDS and Society Research Unit, University of Cape Town, the activist Richard Jeffreys of the Treatment Action Group, the lawyer Jeanne Bergman of HealthGAP, and another activist, Gregg Gonsalves of the AIDS and Rights Alliance for Southern Africa (but still listed as developing the website in March 2006). More people who had not themselves done any actual research in this field.

By 27 January 2008, the “AIDS Truth team members” were listed as “(in alphabetical order):
· Dr. Nicholas Bennett, Department of Pediatrics, University Hospital, Syracuse, New York
· Dr. Jeanne Bergman, The Center for HIV Law and Policy in New York City
· Martin Delaney, Founding director of Project Inform
· Dr. Brian Foley, Los Alamos National Laboratory, Los Alamos, New Mexico
· Bob Funkhouser, Los Alamos National Laboratory, Los Alamos, New Mexico
· Nathan Geffen, Treatment Action Campaign, Cape Town, South Africa
· Gregg Gonsalves, AIDS and Rights Alliance for Southern Africa
· Dr. Bette Korber, Los Alamos National Laboratory, Los Alamos, New Mexico
· Dr. John P. Moore, Weill Medical College of Cornell University, New York City
· Dr. Nicoli Nattrass, Director of the AIDS and Society Research Unit, University of Cape Town, South Africa
· Ken Witwer, Johns Hopkins University, Baltimore, Maryland”

By 26 July 2009, designated as “past contributors” were Delaney (deceased), Jeffreys, Funkhouser, and Moore. Added was Eduard Grebe (AIDS and Society Research Unit, University of Cape Town, South Africa).

By 26 January 2010, Funkhouser was again a team member, and this remains the team except that former graduate student Witwer is now Dr. Witwer.


Posted in antiretroviral drugs, clinical trials, experts, HIV does not cause AIDS, HIV skepticism, Legal aspects | Tagged: , , , , | 3 Comments »

“HIV-positive” and healthy without antiretroviral drugs

Posted by Henry Bauer on 2014/05/30


A film by Andi Reiss and Joan Shenton

Positive Hell is the story of five individuals who have defied their doctors and lived on for nearly thirty years with a diagnosis of death. The film highlights a network of people diagnosed HIV Positive in the province of Galicia in Northern Spain.

Some of them, like physician Dr Manuel Garrido, have never taken any antiviral drugs. He’s been swimming against the tide of medical orthodoxy for three decades.

Raquel has had two children who are fit and well and are HIV negative and her husband, Pablo is also HIV negative.

Others like Manoel took antivirals for a while and found they made him feel so ill he stopped taking them. How can this be? Haven’t we been told that everybody who tests positive is sure to die? Dothese people have a special magic gene that protects them against HIV? Or could it be that this death sentence has been mistaken all along?

The five protagonists describe their struggle to survive when faced with a death sentence, their experiences as social pariahs, their battles with doctors and the medical orthodoxy and their absolute conviction that the science behind AIDS is cruelly wrong.

Posted in Alternative AIDS treatments, antiretroviral drugs, HIV does not cause AIDS | Tagged: , , , | 7 Comments »

AIDS and Idiopathic CD4-T-cell Lymphopenia

Posted by Henry Bauer on 2014/05/11

When “HIV-positive” gay men who have long been healthy then become sick with an “AIDS-like” illness, perhaps they are experiencing idiopathic CD4-T-cell lymphopenia and not “HIV/AIDS”.

* * * * * * * *

The “AIDS” era had begun as GRID: gay-related immunedeficiency. As John Lauritsen pointed out as early as 1985, that naming stemmed from a bizarre, unique, misleading classification scheme which masked the fact that the prime risk factor for illness was not being gay, the prime risk factor was abusing drugs as part of a blatantly unhealthy lifestyle [1].
When Africans, Haitians, and drug injectors were seen to have the same illnesses, GRID was re-named AIDS: acquired immune deficiency syndrome of unknown cause, characterized by low CD4 counts. In 1984, the cause of this immune deficiency was prematurely and mistakenly attributed to a retrovirus claimed to have been discovered by Robert Gallo who subsequently reported detecting “HTLV-III” in “18 of 21 patients with pre-AIDS … [and] 26 of 72 adult and juvenile patients with AIDS” [2], a rate of detection that is anything but convincing proof of cause. The evidence has continued to accumulate  that this “HTLV-III”, subsequently re-named “HIV”, was and is not the cause of “AIDS”, which therefore remains an immune deficiency of unknown or unspecified cause.
By 1993 so many cases of “HIV-negative AIDS” had come to official attention that these were attributed to yet another newly minted condition: Idiopathic CD4-T-cell Lymphopenia, ICL or ITCL: immune deficiency of unknown cause associated with low CD4 counts.

But this is precisely the original definition of AIDS before the red herring of “HIV”. Therefore AIDS Rethinkers and HIV Skeptics, given their understanding that “HIV” does not cause “AIDS”, might logically conclude that “AIDS” is indistinguishable from ICL/ITCL.

* * * * * * * *

What really matters, of course, is not names but illnesses and deaths, and the reasons for those have been thoroughly confused and obscured by several official re-definitions of “AIDS” and by corollaries of the mistaken treatment of “HIV-positive” individuals with toxic “antiretroviral drugs” instead of treatment of manifest illnesses and symptoms, typically opportunistic infections.

None of those manifest illnesses were new or unique to “AIDS”. They were brought about in different ways in the four original (early 1980s) risk groups:
(1) In drug abusers, immune deficiency is brought about directly by the drugs, resulting in “AIDS”-like diseases that were noted already in the 1960s [3].
(2) In Africans, immune deficiency is brought about by malnutrition and periodic infections by a multitude of endemic diseases [4].
(3) Haitians were soon removed from the list of risk groups to which they had been added prematurely.
(4) In gay men — in only a small proportion of gay men, it is not emphasized often enough — immune deficiency had been brought about by drug abuse as part of a blatantly unhealthy lifestyle [1, 5]. The only “AIDS disease” unique to gay men was identified, also mistakenly, as Kaposi’s sarcoma; it was not actually a sarcoma, it was damage to blood vessels caused by ingestion of volatile nitrites, “poppers” [6].

In absence of any known ways to strengthen immune systems, early cases of AIDS among gay men were handled by treating, or by prophylactic measures against, the specific opportunistic infections. Following the mistaken attribution of AIDS to HIV, about 150,000 deaths were then brought about by administration of the supposedly “antiretroviral” AZT [7]. After the replacement of monotherapy by less toxic combinations of AZT-like and other types of drugs (cART or HAART), mortality declined, but it seems to have leveled off since about 1998 at roughly 15,000 annually in the USA [8] (15,529 in 2010 [9]). How much of that mortality is attributable to the drugs cannot be estimated because too many pertinent data are lacking (mortality as a function of length of time on antiretroviral drugs, type of drugs, specific causes of death).

* * * * * * * *

This factual history is a preamble to further consideration of the conundrum addressed in two recent posts [10]: the puzzle of delayed illness in “HIV-positive” gay men. Commentaries to those postings made me realize that “AIDS” is really ICL/ITCL. When “HIV-positive” gay men who have been healthy for long periods without antiretroviral treatment then become ill and have low CD4 counts, the cause cannot be “HIV” because “HIV” does not cause illness and may not even exist; therefore the cause of “AIDS-like” illness must be idiopathic CD4-T-cell lymphopenia. Hence it is worth considering what mainstream science has to say about that condition, two decades after its discovery. If successful treatments for ICL had been found, perhaps those could also work for cases of delayed illness in “HIV-positive” gay men. Unfortunately, I have not come across reports of successful treatments for ICL. Nevertheless, some aspects of ICL may allow for a different approach to cases of delayed illness in “HIV-positive” gay men, in particular recognition of the wide range of possibly associated manifest illnesses, the good probability of recovery from those illnesses, and the occasional observation that ICL reverses itself — CD4 counts sometimes increase, for as mysterious a reason as that for their initial decline.

There are no reliable data about the population-wide prevalence of ICL, because counting CD4 is not part of routine testing. Therefore almost all cases of ICL have been identified following manifest illness later diagnosed as brought on by CD4-specific immune deficiency. Thus it is not known how many people with “low” CD4 counts never become ill. What is known, though, is that CD4 counts in themselves are not valid markers of good or bad health [11].
So when “HIV-positive” gay men who have long been healthy then become sick with an “AIDS-like” illness, not only is “HIV-positive” a red herring, so too is CD4 count. No doubt something specific is at the root of the newly experienced illness, but it isn’t “HIV” and it may not have anything to do with the CD4 level, and it may not be the same “something” in all such cases.

Perhaps most striking in the literature on ICL is the lack of regularities — other than that there is no identified cause for the low CD4-count. A recent review of 258 cases from all over the world [12] found that ICL had been diagnosed at ages ranging from 1 to 85, with a very broad distribution (standard deviation of 19 on a mean of 41). As would be expected from the manner in which cases typically come to light, most had infections “including AIDS-defining illnesses”, but about 1 in 8 were healthy despite low CD4 count. Where illness was present, it ranged over various kinds of mycobacterial and cryptococcal infections as well as candidal and herpes (Varicella zoster); nearly 1 in 5 had a malignancy, and 15% had some sort of autoimmune disease. Only 4 patients had family members who also had low CD4 counts.
Etiology (cause) of ICL remains mysterious, but it has been “hypothesized that abnormally increased microbial translocation through the intestinal wall may be an underlying etiology” — consistent with the intestinal dysbiosis hypothesis in “HIV/AIDS” [13]. Low CD4 counts can also be caused by the manifest infections themselves.
It is encouraging that nearly 4 out of 5 of the described cases survived the manifest illness [12] (but for how long was not reported). Furthermore, “Transient CD4 lymphocytopenia is common and has been estimated to occur in healthy HIV-negative individuals . . . [at rates of] from 0.4-4.1%” [emphasis added], consistent with the literature that reveals CD4 and health to be independent of one another [11]. According to one report, CD4 counts normalized spontaneously in 7 of 39 ICL patients after an average of 31 months [14]. It is further reassuring though puzzling that “Patients with cryptococcal infection and ICL have . . . favorable prognosis . . . [despite] an increased likelihood of developing dermatomal zoster” [15].
There is no agreed general way to treat ICL, but some data “support IL-2 as a relatively safe and potentially effective treatment for ICL patients with opportunistic infections, especially when combined with conventional treatment regimens” [12].
“Adult onset” immunodeficiency [16] may be akin to ICL in some respects, but it is not associated with low CD4 counts. Instead, the opportunistic infections were associated with the presence of anti-interferon-γ auto-antibodies, and a hereditary or genetic predisposition was suggested. Since autoimmune conditions were also found in about 15% of ICL cases [12], it would seem reasonable to look into possible autoimmune conditions whenever ICL is diagnosed or suspected — including cases of “HIV-positive” gay men who have long been healthy but then become ill.

Although ICL is said to be rare, this may only be because little population-wide data on CD4 counts are available and ICL is only identified in the presence of manifest illness. It also complicates matters that “ICL lymphopenia” might actually be owing to the manifest illnesses. Similarly, cases of “HIV-positive” gay men who have long been healthy but then become ill may seem to be rare just because they become known only serendipitously through anecdotal reports when the affected individuals choose to bring their situation to wider attention.
Note that many or most of the manifest diseases associated with ICL are or could be classified as “AIDS-defining”. Therefore an “HIV-positive” gay man who has long been healthy and who then “acquires” ICL — cause unknown — would naturally suspect that HIV/AIDS theory might be correct after all, and he might infer that “HIV” had been long latent and was now making him ill, and he would likely then experiment with antiretroviral drugs. Short courses of antiretroviral drugs have indeed been effective in some instances, but an alternative might be to experiment with speculative possible treatments for ICL: IL-2, probiotics, perhaps steroids which can help against autoimmune diseases, for example Hashimoto’s encephalopathy [17].
In any case, it is worth knowing that nearly 80% of ICL patients recovered from their manifest illness and that in some cases the low CD4 count reversed itself after as much as 3 years.


[1] John Lauritsen, “CDC’s tables obscure AIDS-drug connection”, reprinted as chapter I in The AIDS War (ASKLEPIOS, 1993)
[2] Gallo et al.,. “Frequent detection and isolation of cytopathic retroviruses (HTLV-III) from patients with AIDS and at risk for AIDS”, Science, 224 (1984) 500-3
[3] P. 103 f. in Neville Hodgkinson, AIDS: The Failure of Contemporary Science, 1996.
[4] Robert S. Root-Bernstein, Rethinking AIDS — The Tragic Cost of Premature Consensus, 1993; especially chapter 9
[5] For descriptions of that “fast-lane’ life-style, see the sources cited in section of The Case against HIV
[6] John Lauritsen & Hank Wilson, Death Rush: Poppers and AIDS, 1986
[7] Section 5.1.5 of The Case against HIV
[8] Figure 3, p. 21 in Deaths Among Persons with AIDS through December 2006, HIV/AIDS Supplemental Report, 14 #3
[9] Statistics Overview, “Deaths of persons with an AIDS diagnosis”, updated 23 April 2013;
[10] The Lazarus Effect in HIV/AIDS; The Lazarus Effect and the puzzle of delayed illness in “HIV-positive” gay men
[11] David Crowe, “Predictability of a CD4 count”, July 2012;
[12] Dina S. Ahmad, Mohammad Esmadi, and William C. Steinmann, “Idiopathic CD4 Lymphocytopenia: Spectrum of opportunistic infections, malignancies, and autoimmune diseases”, Avicenna Journal of Medicine, 3 (#2, April-June 2013): 37-47
[13] See sections & of The Case against HIV
[14] Zonios et al., “Idiopathic CD4 lymphocytopenia: natural history and prognostic factors”, Blood, 112 (2008) 287-94)
[15] Zonios et al., “Cryptococcosis and idiopathic CD4 lymphocytopenia”, Medicine (Baltimore), 86 (#2, 2007) 78-92
[16] S. K. Browne et al., “Adult-Onset Immunodeficiency in Thailand and Taiwan”, New England Journal of Medicine, 367 (2012) 725-34
[17] When doctors can’t tell you what’s wrong (updated); Encephalitis and Hashimoto’s Encephalitis


Posted in Alternative AIDS treatments, antiretroviral drugs, HIV does not cause AIDS, HIV risk groups, HIV skepticism | Tagged: , | 3 Comments »


Get every new post delivered to your Inbox.

Join 119 other followers