HIV/AIDS Skepticism

Pointing to evidence that HIV is not the necessary and sufficient cause of AIDS

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About money, and HIV in Estonia

Posted by Henry Bauer on 2016/02/12

A correspondent from Estonia shared this recent news:

Unique HIV is spreading in Estonia
Research has shown that recombined (?) [recombinant = hybrids of the known “strains”] form of HIV spreads in Estonia. People get infected locally and it is not brought in from abroad.
‘We have done more than 10 years of research and we do not have exporters [?importers?] of HIV, we do not get it from Russia or Western Europe’, says Irja Lutsar, professor of medical microbiology and virology. She added that people get infected with HIV locally and a recombined [recombinant] form of HIV is spreading here.
‘Our virus is unique but if you ask where it came from then I do not know answer to that.’”

Professor Lutsar might find some clues in de Harven’s article in Journal of the American Physicians and Surgeons  as well as in The Case against HIV.

“Also, US, British and Finnish embassies here recently wrote a public letter to Estonian parliament about HIV”.

Part of that letter reads;
“The U.S. President’s Emergency Plan for AIDS Relief (PEPFAR) is the largest commitment by any nation to combat a single disease. Through PEPFAR, the U.S. government works with vulnerable, developing nations on a framework to combat HIV/AIDS. Since 2009, the U.S. government has committed nearly $65 billion to support PEPFAR and its global HIV/AIDS response. While gains have been real, progress against this disease, even in the United States, has been uneven.”

That inevitably brought to mind the quote attributed to the late Republican fiscal conservative, Senator Everett Dirksen:

“A billion here, a billion there, pretty soon, you’re talking real money”.

(As commonly with the most delightful quotes, this may be a mis-attribution)

At any rate, the United States has apparently disbursed $65 billion without anyone getting any benefit and some undoubtedly being harmed as a result of being fed toxic drugs while not only healthy but even HIV-negative.
I shouldn’t have said, of course, that no one benefited. The drug companies and their shareholders and executives have certainly benefited.

 

Posted in antiretroviral drugs, Funds for HIV/AIDS, HIV absurdities, HIV risk groups, HIV skepticism, HIV tests, HIV/AIDS numbers, uncritical media | Tagged: , , | 7 Comments »

Who can be trusted about science? Not the Royal Society of London or the National Academy of the United States

Posted by Henry Bauer on 2016/01/02

Those of us who have bothered to look into the evidence about HIV and about AIDS know that the mainstream dogma, that HIV causes AIDS, has no credible evidence in its favor; and that prestigious and authoritative institutions and organizations persistently disseminate the false belief and try to suppress the evidence and those who present it. The most obvious disproof of HIV=AIDS is that HIV is incapable of causing any sort of epidemic because it is not sufficiently transmissible, less than 1 per 1000 acts of unprotected intercourse (and only 1.4 per 1000 acts of unprotected receptive anal intercourse, supposedly the most risky).

At the Rethinking AIDS Conference in Oakland,  I had pointed out that this resistance to contrary evidence is unfortunately not uncommon, it is evident on a variety of topics; see also my book, Dogmatism in Science and Medicine: How Dominant Theories Monopolize Research and Stifle the Search for Truth (McFarland 2012).

One topic on which mainstream dogmatism contrary to evidence is most pronounced is global warming and climate change: do human activities contribute appreciably to these phenomena, outweighing natural forces? The evidence says no, or at least there is no empirical proof of it. Nevertheless, all mainstream groups insist that the science is settled — and that includes the Royal Society of London and the National Academy of Science of the United States, which mislead disgracefully on this topic.

 

Posted in experts, uncritical media | Tagged: , , , | 18 Comments »

Additions to Case against HIV

Posted by Henry Bauer on 2015/10/20

Lukas’s comments have been added to The Case against HIV

5.3.1.3 Risk of non-AIDS-related mortality may exceed risk of AIDS-related mortality among individuals enrolling into care with CD4+ counts greater than 200 cells/mm3
915.

5.3.1.4 Liver fibrosis caused by some ARVs
916.

Posted in antiretroviral drugs | Tagged: | 10 Comments »

New dissident book

Posted by Henry Bauer on 2015/10/18

Le Falsità sull’AIDS
Ancora Imbrogliati Dalla Scienza? d  by Domenico Mastrangelo

Google can translate the pages (sort of), enough to be able to get a sense of teh book from the translated Forewords

Posted in HIV does not cause AIDS | Tagged: , | 7 Comments »

Gay genes and HIV

Posted by Henry Bauer on 2015/10/06

Some 20 years ago, Dean Hamer reported an association between certain DNA markers and being gay [1]. The report was met with considerable skepticism. Now a new study [2] has reached much the same conclusion as Hamer. This may be relevant to the apparently greater frequency of “HIV-positive” among gay men.

Overall data are clear, that “HIV-positive” does not behave like an infectious condition [3]. More specifically, if “HIV-positive” is ever transmitted sexually then it is with essentially negligible probability, according to the Centers for Disease Control & Prevention:

Heterosexual vaginal transmission is estimated as less than 1 per 1000, but receptive anal intercourse is estimated at 1.4%. This is still less by a large factor than the transmissibility of known venereal diseases like syphilis and gonorrhea. Where does the estimate originate?

It cannot be based on observations in prisons since several such studies reported much lower rates there (p. 47 in [3]). Rather, the estimate likely comes from data on “HIV-positive” among gay men who frequently practice receptive intercourse. In other words, there is a correlation between being gay, receptive anal practices, and testing “HIV-positive”. In prisons, there is a significant amount of anal intercourse by men who are not gay, yet this apparently does not correlate with becoming “HIV-positive”. Evidently it is being gay, more than anal intercourse, that correlates with being “HIV-positive”.

If there is a genetic pre-disposition to being gay, as the Hamer and Sanders studies indicate, then perhaps there is also a genetic pre-disposition among gay men to testing “HIV-positive”.

That some genetic characteristics do predispose to testing “HIV-positive” is demonstrated by racial differences. Men of sub-Saharan ancestry test “HIV-positive” at rates about 7 or 8 times greater than with Caucasian men and about 10 times greater than with Asian men. There are also racial differences in the sensitivity of “HIV” tests to the p24 protein which is one of the “HIV” markers (section 3.4 in The Case against HIV).

I’m not suggesting, of course, that genes could be the sole reason why gay men are more frequently “HIV-positive” than others. Genetic pre-dispositions are probabilistic. Not all gay men test “HIV-positive”. In the earliest days of AIDS, only a small proportion of gay men became ill. Many gay men are both “HIV-positive” and healthy and never contract “AIDS”-type diseases.
Moreover, “HIV-positive” reflects any number of possible conditions, most of which are experienced equally by gay men and everyone else (section 3.2.2 in The Case against HIV).

Similarly, the Hamer and Sanders studies do not suggest that genetics determines sexual orientation, merely that it can bring a heightened tendency; it is explicitly a small effect, to the degree that genetic studies on infants or embryos could not have any useful predictive value [2]. It is widely agreed that behavioral characteristics in general arise from some combination of hereditary and environmental factors. Moreover, it remains to compare the frequent correlation of certain genetic factors with being gay to the overall frequency of those particular factors among all men, which would indicate how strongly those factors may predispose toward a preferred sexual orientation.

So explanations for the greater incidence of “HIV-positives” among gay men are obviously and necessarily partial and multiple. I believe that some proportion of “HIV-positives” among gay men, correlated with also becoming ill, can be explained by the intestinal dysbiosis theory. Here I am suggesting that one possible and additional reason why some gay men are “HIV-positive” may be a genetic pre-disposition, particularly when “HIV-positive” does not correlate with a high probability of illness. Since the markers identified by Hamer and Sanders are not exclusive to gay men, a linkage between those markers and testing “HIV-positive” could also explain some of the incidence of “HIV-positive” among men who are not gay.
————————————
[1] Dean H. Hamer et al., “A linkage between DNA markers on the X chromosome and male sexual orientation,” Science 261 (1993) 321-7
[2] A. R. Sanders et al., “Genome-wide scan demonstrates significant linkage for male sexual orientation”, Psychological Medicine 45 (2015) 1379-88
[3] Henry H. Bauer, The Origin, Persistence and Failings of HIV/AIDS Theory, McFarland 2007

 

Posted in clinical trials, HIV and race, HIV risk groups, HIV tests, HIV transmission, HIV/AIDS numbers, sexual transmission | Tagged: , | Leave a Comment »

 
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