People carrying black-African genes test “HIV-positive” at far greater rates than do people without that genetic ancestry. HIV/AIDS theory “explains” that by postulating greater rates of careless “not-safe-sex” promiscuity and infected-needle-sharing drug injection. Thereby HIV/AIDS theory postulates significant genetic determination of behavior, which in other contexts is dismissed as pseudo-science.
Moreover, actual observations and studies have repeatedly shown that the facts vitiate that proposed “explanation”: Africans and African-Americans indulge in risky behavior at lower rates than do white Americans (pp. 77-9 in The Origin, Persistence and Failings of HIV/AIDS Theory).
The conclusion is inescapable: HIV/AIDS theory is radically wrong about how “HIV-positive” is transmitted.
But that inescapable conclusion continues to escape mainstream practitioners and researchers and such media pundits as Donald G. McNeil Jr. of the New York Times (Poor Black and Hispanic men are the face of H.I.V.):
“The AIDS epidemic in America is rapidly becoming concentrated among poor, young black and Hispanic men who have sex with men”
NO. There’s nothing recent or rapid about it. The racial disparities have always been there (Chapters 5 & 6 in The Origin, Persistence and Failings of HIV/AIDS Theory).
Furthermore, it is black WOMEN who are most affected compared to others, 20 times more likely to be “HIV-positive” than white women, whereas for males the ratio is (“only”) 7.
“Nationally, 25 percent of new infections are in black and Hispanic men, and in New York City it is 45 percent”
Yes, of course, because it’s blackness that contributes overwhelmingly to testing “HIV-positive”. Hispanics in New York are primarily of black Caribbean-African stock, whereas West-Coast Hispanics are largely non-black, of Latin-American stock. Therefore national-average rates of “HIV-positive” among Hispanics are lower than East-Coast Hispanic rates of “HIV-positive” (pp. 57-8, 71-2 in The Origin, Persistence and Failings of HIV/AIDS Theory).
“Nationally, when only men under 25 infected through gay sex are counted, 80 percent are black or Hispanic — even though they engage in less high-risk behavior than their white peers” [emphasis added]; “a male-male sex act for a young black American is eight times as likely to end in H.I.V. infection as it is for his white peers. That is true even though, on average, black youths in the study took fewer risks than their white peers: they had fewer partners, engaged in fewer acts of sex while drunk or high, and used condoms more often”.
So McNeil is even aware of this conundrum which falsifies the central axiom of HIV/AIDS theory, namely, that HIV is transmitted as a result of risky behavior. Yet he does not follow this statement of fact with any explanation of this paradox which contradicts and falsifies mainstream views.
Instead, McNeil passes on without comment the usual meaningless weasel-words about some unspecified “intervention”:
“Critics say little is being done to save this group, and none of it with any great urgency. ‘There wasn’t even an ad campaign aimed at young black men until last year — what’s that about?’. Phill Wilson, president of the Black AIDS Institute in Los Angeles, said there were ‘no models out there right now for reaching these men’”.
What conceivable use could any models be, when it’s acknowledged that these supposedly at-high-risk people already practice less risky behavior than the no-high-risk white folk?
Still, of course there’s no harm in asking for more money even in absence of any clue what to do with it:
“With more resources, we could make bigger strides”.
What the mainstream says about the high rates of black “HIV-positives” is pitifully, woefully inadequate; it misses the whole point. It suggests that although their behavior is less risky, black folk have “other risk factors. Lacking health insurance, they were less likely to have seen doctors regularly and more likely to have syphilis, which creates a path for H.I.V.”
But it’s yet another counterfactual canard that syphilis and other STDs make it more likely that someone will “contract” “HIV”, i.e. become “HIV-positive”: there is simply no correlation between incidence of STDs and of “HIV” (pp. 31-5, 109 in The Origin, Persistence and Failings of HIV/AIDS Theory).
As to insurance, what is the evidence that having health insurance makes for lower rates of being or becoming “HIV-positive”? This is simply hand-waving bullshit* emitted because no sensible explanation can be offered.
As to seeing doctors regularly, what is the evidence that seeing doctors regularly makes for lower rates of being or becoming “HIV-positive”? Quite the opposite, in fact: The largely white gay men who first contracted “AIDS” had mostly been seeing doctors very often because of their constant need for treatment after suffering all sorts of illnesses. Dr. Joseph Sonnabend, with a practice of largely gay clients in New York in the 1970s, had in fact warned his regular customers that if they did not change their lifestyle something drastic and awful would befall them.
And then, “Other risk factors include depression and fatalism” — What, pray, is the mechanism by which those conditions produce “HIV-positive”? Among people who are acknowledged to behave less riskily than those who are not at high risk of becoming “HIV-positive”?
Another popular non-explanation is that blacks become “HIV-positive” more often because “HIV-positive” is so much more common in the black community: It’s more common because it’s more common.
I cannot imagine a higher degree of hypocrisy, intellectual vapidity, sheer unwillingness to draw obvious conclusions from undisputed facts, than is demonstrated without fail and without end by mainstream researchers, doctors, and pundits when confronted with the plain fact that blackness makes for being “HIV-positive”.
Not that this perverse behavior is much different from behaving as though testing “HIV-positive” proved infection by “HIV” when standard authorities have long stated quite forthrightly that there is no gold standard “HIV” test, no test capable of demonstrating actual infection by “HIV”, and that the rates of false positives are inevitably high (Stanley H. Weiss & Elliot P. Cowan, “Laboratory detection of human retroviral infection”, chapter 8 in Gary P. Wormser (ed.), AIDS and Other Manifestations of HIV Infection, 2004 (4th ed.).
No technical expertise is needed to recognize the sheer unadulterated nonsense of talking about “risk factors” when the known end-result is less risky behavior. How can any number of purported risk factors be alleged to heighten risk when the facts show that the risk is lower of the only behavior that supposedly transmits “HIV”?
* Words uttered without regard to their truth — Harry Frankfurt, On Bullshit, Princeton University Press, 2005.