HIV/AIDS Skepticism

Pointing to evidence that HIV is not the necessary and sufficient cause of AIDS

Posts Tagged ‘Kyrgyzstan’

HIV/AIDS in Italy — and “NEEDLE ZERO”

Posted by Henry Bauer on 2008/10/11

Professor Marco Ruggiero, University of Florence (Italy) kindly forwarded a copy of a PhD thesis presented on October 8. He tells me that it is now “freely available for consultation in the Library of the Department of Experimental Pathology and Oncology of the University of Firenze, Italy (”; the citation is

Scarpelli S. “HIV infection and AIDS in Italy: results supporting the chemical hypothesis”.
PhD Thesis in Biological Sciences, Faculty of Mathematical, Physical and Natural Sciences, University of Firenze, Italy, October 8, 2008. (

I can’t read Italian, but the thesis has an Abstract in English with some fascinating information:

There is no “Italian registry of HIV cases; there are no data concerning the number of new HIV infections in Italy”. The Ministry of Health does issue estimates, but “the lack of data does not allow to support the statement that there is (or that there has ever been) a HIV/AIDS epidemic in Italy; neither it allows to establish whether HIV is the cause of AIDS in Italy. This regrettable absence of surveillance is due, among other considerations, to the so called Privacy Law that, should AIDS be caused by HIV, evidently protects the individual’s right to privacy more than public health. Thus, if a laboratory finds out that an individual is HIV-positive, this information cannot be disclosed to anybody but the individual, who is then free to disregard the information and spread the virus. In fact, the Law states ‘L’identificazione del malato di HIV deve essere effettuata con modalità che non consentano l’identificazione della persona’ (art. 5, comma 2, l n. 135/1990), i.e. ‘identification of the HIV patient has to be performed with modalities that do not allow identification of the person’.”

I was struck particularly by the official recognition that HIV/AIDS is not a threat to public health. AIDS (not HIV infection) is classified “only as a third class [least dangerous or harmful] disease”, whereas influenza is in the first class and hepatitis (A, B, and C) are in the second.

Simone Scarpelli “tested the chemical hypothesis by analysing the data obtained by the rehabilitation centres for drug abuses (SerT, Servizi per le Tossicodipendenze). The data show that there is a good correlation between recreational drug abuse and AIDS cases in Italy.”

While the rate of heroin confiscation has not varied much, the pattern of consumption has changed from high usage by relatively few addicts to lower average use by a larger number of people who do not regard themselves as addicted and don’t seek treatment. The data are consistent with “a linear-quadratic model for heroin effects on the immune system and the development of AIDS” similar to that for “the biological effects of ionizing radiations and it could explain the bell-shaped curve of AIDS, the flat curve of heroin confiscation and the decreasing curve of heroin addiction in Italy. In fact, at high doses (such as in the eighties and the early nineties) the effects of heroin on the immune system are deterministic and drug addicts developed AIDS; at lower doses, however, the effects are stochastic i.e. there is only an increased probability of impairing the immune system and this might account for the decreasing AIDS incidence. According to this interpretation of the only available data for Italy, the AIDS epidemic paralleled the severe heroin abuse of the past. Nothing could be said about HIV since no data are available. This interpretation is also consistent with the recent meta-analyses that demonstrate the failure of anti-retroviral drugs in increasing survival of HIV-positive subjects (Lancet 2006; 368: 451-58), and with the statement that an AIDS vaccine could never exist (N. Engl. J. Med. 2007; 357: 2653-55).”

Scarpelli’s work supports Duesberg’s “drug-AIDS hypothesis”, for which massive evidence is collected in Duesberg, P., Koehnlein, C. and Rasnick, D., “The Chemical Bases of the Various AIDS Epidemics: Recreational Drugs, Anti-viral Chemotherapy and Malnutrition”, J. Biosci. 28 [2003] 383-412.


In the early days of “AIDS”, a certain airline steward was identified as the “Patient Zero” whose profligate promiscuity supposedly seeded AIDS around the USA. That story is inconsistent with the current belief that illness follows infection only after an average interval of about 10 years, for the claimed victims of Patient Zero’s exploits became ill within months of their contact with him, that’s how they could be identified or traced — see Shilts, And the Band Played On: p. 130, “long latency period” of 10 and 13 months in two cases. I’m not aware that this inconsistency has been remarked on in mainstream discussions, any more than the myriad other facts inconsistent with HIV/AIDS theory. I mention Patient Zero because he exemplifies the mystery of the origin of the supposed HIV/AIDS epidemics — most particularly, perhaps, those epidemics supposedly spread primarily by the sharing of needles. How does such an epidemic get started, let alone continue to spread?

Recall the authoritative recent review that I described as a textbook instance of cognitive dissonance, “The spread, treatment, and prevention of HIV-1: evolution of a global pandemic”, by Myron S. Cohen, Nick Hellmann, Jay A. Levy, Kevin DeCock, and Joep Lange, Journal of Clinical Investigation, 118 [2008] 1244-54; doi:10.1172/JCI34706, whose authors are heavyweight mainstream HIV/AIDS gurus — Levy and  DeCock have been in this business from the beginning, though DeCock blotted his copybook somewhat by admitting that there had not been and never would be heterosexual epidemics outside Africa — “WHO Says That We’ve Been Very Wrong about HIV and AIDS? (Clue: WHO = World Health Organization)”, 10 June 2008.

According to that authoritative review, different regions of the globe see HIV spreading by dramatically different pathways:

Figure A

“The HIV-1 epidemic in Western Europe is diverse but was initially fueled by infections among MSM and injecting drug users, the latter especially in the southern part of the continent (3). Italy, Spain, Portugal, France, and the United Kingdom have been most heavily affected (3). Heterosexual transmission of HIV-1 in Europe has slowly increased, and many infections today are found among immigrants from sub-Saharan Africa (3). In Eastern Europe, where brisk and severe epidemics emerged among injecting drug users in the late 1990s, the most affected countries are the Russian Federation and Ukraine (3)” — (3) is UNAIDS, “AIDS epidemic update: December 2007”.

Now, the postulated “HIV” can’t survive for long outside bodily fluids, so the needle that supposedly transfers it must have been wetted and “infected” not much earlier. Try to construct a scenario in which that’s compatible with the regional situations in Figure A. Let’s say an infected male, Patient One — gay, bisexual, or heterosexual — enters Eastern Europe and infects a drug addict; whereupon the “virus” spreads like wildfire via the necessarily postulated orgies of needle sharing, but the infection doesn’t spread much to people who just have sex without sharing needles. What happened to Patient One? Did he leave the country again? Or did he become much less inclined to have sex, at least with people who are not needle-sharing addicts?

The absurdity is illustrated by several stories from Kyrgyzstan. “According to the CIA Fact Book, by 2003 there were in Kyrgyzstan an estimated 3900 people living with HIV/AIDS, there had been fewer than 200 HIV/AIDS deaths, and the prevalence was estimated at < 0.1% (as low as anywhere in the world)” — “SMART” Study Begets More Cognitive Dissonance, 11 June 2008. In that land where HIV is so rare, “’at least 26 people, mostly children, [were] infected in two local hospitals’. . . and medical personnel were fired” [HIV-Positive Children, HIV-Negative Mothers, 25 November 2007] because, obviously, these HIV-positive children of HIV-negative mothers could only have become HIV-positive via infected needles. How did those needles become infected in the first place? Of necessity, not long before the babies were supposedly stuck with them . . . . Were the babies all injected with the same dirty needle in rapid succession, or were there 26 different sources of infection, each of them contributing a dirty needle just in time for a baby to get stuck immediately thereafter?

See also “Babies Infect Mothers; Crazy Theory Ruins Lives”, 12 April 2008: Those babies were then apparently capable of infecting their mothers as they suckled — and this in Kyrgyzstan, which doesn’t have the vampire tradition of Transylvania — or, at least, there have so far been no reports of baby vampires in Kyrgyzstan, only a wild woman or perhaps a monkey  [Kyrgyzsylvania,  Thursday, June 19, 2008].
Of course, if it was a monkey, then the source of HIV in Kyrgyzstan becomes immediately obvious — it’s an African monkey of the ilk that first infected humans with HIV decades ago (supposedly in the knee of Africa, where there’s not nearly as much “HIV” as in southern Africa, where “HIV” is rampant — Deconstructing HIV/AIDS in “Sub-Saharan Africa” and “The Caribbean”, 21 April 2008 ).

Posted in HIV absurdities, HIV in children, HIV risk groups, HIV transmission, HIV/AIDS numbers, sexual transmission | Tagged: , , , , , , , , , , , | 7 Comments »


Posted by Henry Bauer on 2008/06/06

HIV/AIDS has an unparalleled ability to generate grants and gifts.

The Global Fund has approved nearly $29 million, and actually disbursed already $16 million, to help fight HIV/AIDS in the hard-hit land of Kyrgyzstan.

According to the CIA Fact Book, by 2003 there were in Kyrgyzstan an estimated 3900 people living with HIV/AIDS, there had been fewer than 200 HIV/AIDS deaths, and the prevalence was estimated at < 0.1% (as low as anywhere in the world). So the Global Fund’s allocation to Kyrgyzstan, at $29 million, represents about $150,000 per death and about $7000 per patient.

While that may seem excessively generous, perhaps it was guided by the budgeting of the National Institutes of Health, which called in 2007 for about $180,000 per AIDS death in the United States (allocations for other diseases were, for example, just under $10,000 per cancer death and $2600 per cardiovascular death—see STOPPING THE HIV/AIDS BANDWAGON—-Part II, 1 February 2008.

The threat to Kyrgyzstan from HIV/AIDS is further illustrated by the suspected infection of 26 babies in two hospitals (HIV-POSITIVE CHILDREN, HIV-NEGATIVE MOTHERS, 25 November 2007) and the even more terrifying fact that these babies might then infect their mothers through being breast-fed (BABIES INFECT MOTHERS; CRAZY THEORY RUINS LIVES, 12 April 2008 ).

Posted in Funds for HIV/AIDS, HIV absurdities, HIV risk groups, HIV transmission, HIV/AIDS numbers | Tagged: | Leave a Comment »


Posted by Henry Bauer on 2008/04/12

What can one say about tragedies like these?

Kyrgyz Babies Pass HIV to Mothers
OSH, Kyrgyzstan (AP) — Not long ago, she was a wife, mother and teacher. Now Dilfuza Mustafakulova is HIV-positive and has lost her husband and her job. Mustafakulova’s baby son was among 72 children infected with the virus at two Kyrgyz hospitals. Sixteen mothers also have contracted it — in some cases by breast-feeding their children. . . .
The scandal has led to charges of negligence against 14 medical workers in the impoverished former Soviet republic, where investigators suspect the children were infected by tainted blood and the reuse of needles. . . . Although HIV infection from breast-feeding is rare, it is possible, usually when the baby has mouth sores and the mother has lesions on her nipples, according to AIDS experts. Mustafakulova, whose son was 7 months old at the time, said her breasts were cracked and bleeding. . . . Some 1,600 people are infected with HIV in the Central Asian nation of 5 million people, according to official figures — 15 times more than in 2002. AIDS experts estimate the real number is closer to 6,000. The majority of cases stem from intravenous drug use. . . .
Mustafakulova’s troubles began in June, when her son developed a high fever. She took him to the Nookat hospital, where she said doctors put him on an intravenous drip. When he did not get better, she took him to the hospital in Osh, the country’s second-largest city. After more than a month in the hospital, her son still was not well and she was also feeling weak, so they returned to their village . . . . In October, they both tested positive for HIV. . . . It has not been established where the infection originated. Of the 72 children infected, some were treated only in Nookat and others only in Osh, so both hospitals are suspected. ‘Where else could my child and I become infected if I don’t use narcotics and don’t live an immoral life?’ Mustafakulova said during a recent visit to the Rainbow center. ‘This could only be the irresponsibility of doctors.’ She was abandoned by her husband . . . .  No longer welcome in her in-laws’ home, she and her children moved in with her parents. She sold her only possession, a small plot of land, to pay for her son’s medical treatment. . . .  The story of Mustafakulova’s fellow villager, Zarifa Shamshiyeva, is remarkably similar. ”


On what evidence do the experts rely for the view that mothers can be infected in this way?

Even the higher estimate of 6000 infected in a country of 5 million makes the rate only 1.2 per 1000, which is typical for low-risk populations in non-African countries where there has been no epidemic during the two decades of the AIDS era, despite continual prediction of such epidemics by “AIDS experts”.

Here’s an assignment:

In 2002, only about 100 people were infected.
Most new infections have come via needles.
Construct a plausible scenario to account for how this mechanism brought a 15-fold increase in infections in half-a-dozen years.


The tragedies here are not only the wrongly diagnosed babies and mothers. What about the doctors and other medical personnel who are being charged with negligence, when they did nothing to bring about this situation?

Four health officials from southern Kyrgyzstan were fired for their alleged roles in the outbreak, including the directors of the two hospitals. The Kyrgyz General Prosecutor’s office has opened a criminal investigation into the incident.”

“Kyrgyz medical workers charged with infecting children with HIV”  [Associated Press, March 20, 2008]
“BISHKEK, Kyrgyzstan: Fourteen health professionals in Kyrgyzstan will face trial for allegedly infecting children with HIV”

Posted in experts, HIV absurdities, HIV in children, HIV transmission, HIV/AIDS numbers, Legal aspects | Tagged: , , , | 23 Comments »


Posted by Henry Bauer on 2007/11/25

Children not infected by their mothers, and not victims of pedophiles, could become HIV-positive only via infected needles or transfused blood, according to the orthodox view of HIV/AIDS. But a number of reported instances cannot plausibly be explained in this fashion. Instead, they support once again the interpretation of “HIV-positive” as a non-specific marker of physiological stress or challenged health.

* * * * * *

Gisselquist recently cited 42 instances of HIV-positive babies born to HIV-negative mother in South Africa (“Not investigating HIV riddles puts lives at risk”, Business Day (Johannesburg), 4 October 2007). He ascribes these infections to unhygienic medical procedures.

In Britain, 5 of 25 mothers of HIV-positive newborns had tested HIV-negative when entering antenatal care (Struik et al., Arch Dis Child., 12 September 2007 [Epub ahead of print] PMID: 17855439). It was speculated that they must have become infected while they were pregnant.

No explanation was offered about the 4-month-old baby in India who was found to be HIV-positive while neither parent, nor the child’s older sibling, was HIV-positive (, accessed 21 December 2007).

Allegations that children became infected with HIV in hospitals or orphanages as a result of unhygienic procedures have also been made in Kazakhstan, Kyrgyzstan, Libya, Romania, and Russia. The Libyan case was widely reported because foreign medical personnel were charged with deliberately infecting children–400 of them in a single hospital (for much detail, see Wikipedia). In Kazakhstan, “at least 78 children have been infected with the HIV virus through the negligence of healthcare workers” (Joanna Lillis, “Government in Kazakhstan Addresses HIV-Infection Scandal” 10/25/06 ); later investigations reported that in 3 hospitals, more than 100 children had become infected in 2006 (cited by Gisselquist, see above). In Kyrgyzstan, “at least 26 people, mostly children, [were] infected in two local hospitals” (Daniel Sershen, “Kyrgyzstan: Officials Grapple with HIV Outbreak”, 10/30/07 ) and medical personnel were fired (“Four more toddlers infected with HIV in outbreak in Kyrgyzstan”,, accessed 26 October 2007). (For further details regarding Kazakhstan and Kyrgyzstan, see the Archives at

Gisselquist (above) describes the following events in Romania and Russia. In Romania, one HIV-positive child of an HIV-negative mother led to further testing, whereupon 12 of 30 children in the same hospital were found to be HIV-positive; widespread testing then found, within a couple of years, 1300 infected–few of them with HIV-positive mothers–among the 12,000 tested. In Russia, it was believed that a single HIV-positive child had led within a couple of years to the infection of 260 children in the same hospital.

* * * * * *

The worldwide consensus over the Libyan affair exonerated the medical personnel from having deliberately infected those 400 children. But how likely is it that these hundreds were all infected accidentally? Could there be so much HIV around in the first place to contaminate the medical instruments? Could the failure to sterilize be really so pervasive? Could conditions have been similarly risky in the hospitals of Kazakhstan, Kyrgyzstan, and Romania, when the prevalence of HIV in those countries is so very low, at ≤0.1% (UNAIDS 2006)? Most of the HIV-positive people in those countries are injecting drug abusers; do hospital personnel perhaps use needles borrowed from drug addicts?

Bear in mind that, no matter what the official propaganda says, the official data make clear that it is extraordinarily difficult to transmit the “HIV-positive” condition via infected needles–see pp. 47-48 of The Origins, Persistence and Failings of HIV/AIDS Theory for citations of the peer-reviewed literature reporting, for example, that “HIV-positive” was 34% among injecting drug users (IDU) who did not share needles and only 19% among those who did; an independent study in Montreal found that clean needles were associated with a ten-fold increase in the odds of seroconverting to HIV-positive; there was no spread of HIV among IDU prisoners in Maryland during 2 years; medical personnel have not contracted HIV or AIDS through needle-stick accidents–the risk was estimated at about 0.3% (whereas for hepatitis the risk is > 10%) and only 57 possible instances had been reported by December 2001, when the count of AIDS cases stood near 800,000.

A large unknown is this: For how long can HIV particles remain infectious outside a living body? Long enough for hundreds of children to have been infected within a few short years? That seems extremely unlikely. But if not dirty needles , then what can explain these epidemics of HIV-positive children?

As already suggested, a ready explanation is that “HIV-positive” is the sign of physiological stress having nothing to do with infection by a human immunedeficiency virus. Strong evidence for this comes from the manner in which HIV-positive varies with age (for further details, see Tables 25-27 and associated text in The Origins, Persistence and Failings of HIV/AIDS Theory). The following schematic diagram, shown also in the post of 18 November,


is based on a large number of individual reports. For ages below the teens, there are four sets of data from public testing sites across the USA (1995-98), one from hospital patients in New Jersey (1988), and one from healthy subjects in Africa (1984-86). Remarkably enough, all showed a similar decrease of the rate of HIV-positives after birth, a decline of about 3/4 in the first year or so. As reflected in the diagram, the rate among newborns was not far from the highest rates recorded at any age, and the lowest rate was in the early teens in all cases.

It seems inconceivable that rates of infection by some contagious agent would show such similar variations with age in such different groups of subjects. On the other hand, this is precisely what one would expect if HIV-positive is a marker of physiological stress. Newborns are immediately challenged to cope with circumstances less friendly than the womb–as noted in an earlier post, Nature has formulated mothers’ milk in a way that helps the infant ward off infections. Over the years, the child’s immune system adapts and the child becomes better able to ward off environmental insults and infections–so, signs of physiological stress become less evident, and the rate of “HIV-positive” declines.

The CDC’s data sets from public testing sites show separately the rates of HIV-positive for females and for males: the latter is greater, by 50% or more. That is again consistent with an explanation in terms of physiological stress, for the natural mortality of male children is higher than that of females. By contrast, it would not be so easy to conjure an explanation of why mothers transmit an infection to male babies 50% more often than to female babies.

Other evidence that HIV-positive marks physiological stress are cited at p. 85 in The Origins, Persistence and Failings of HIV/AIDS Theory, for example: critically ill patients, particularly those in emergency rooms, had higher rates of HIV-positive than others, and unexpectedly high rates of HIV-positive were also found in autopsies.

Once it is accepted that “HIV-positive” is a marker of physiological stress, it becomes rather obvious why it is reported from hospitals in many countries that a significant number of children test HIV-positive even as their parents test negative: the reason is the same as the reason why they are in hospital in the first place, they are experiencing a challenge to health, some degree of physiological stress from any of a variety of possible sources. Surely this is a more plausible line of reasoning than one that has to envisage HIV-infected instruments in large-scale use in several countries, even those where the rate of HIV-positive in the general population is as low as 0.1%; or reasoning that has to envisage that, in Britain, 20% of HIV-positive newborns have that infection because their mothers practiced unsafe sex or drug-injecting even while they were pregnant.

These data about HIV-positive children of HIV-free parents confirms what one can learn from studies of HIV and breast-feeding and from the reports that married women in many places are at the greatest risk for becoming HIV-positive: “HIV-positive” does not signal infection by a deadly virus.

Data about AIDS as well as HIV-positives among children also throws direct doubt on the orthodox view that “HIV-positive” presages progression to AIDS. According to the CDC’s 2005 Surveillance Report, for every 137 adults “living with HIV” in 2005, there were 174.5 “living with AIDS”; among children below 13 years of age, for every 7.4 “living with HIV” there were 2.7 “living with AIDS”. That seems to indicate that the chances of a child progressing from HIV to AIDS is much less than the chance of an adult doing so: for every HIV-positive child, there is only one in three (2.7/7.4 = 0.36) with AIDS, whereas for every HIV-positive adult, there is more than one with AIDS, 137/174.5 = 1.27. Is it conceivable, does it make sense, that children could be 3½ times (1.27/0.36) better able to resist progression to disease than adults?

Posted in HIV does not cause AIDS, HIV in children, HIV transmission, HIV varies with age, M/F ratios | Tagged: , , , , , , , | Leave a Comment »