HIV/AIDS Skepticism

Pointing to evidence that HIV is not the necessary and sufficient cause of AIDS

Archive for the ‘HIV skepticism’ Category

“HIV” is NOT sexually transmitted — yet more clear evidence

Posted by Henry Bauer on 2014/03/27

Recent Nobelist in biology, Randy Schekman, launched a venture to improve publication of valuable research (Science rewards hucksters and spin artists, not soundly tested science): the Open Access on-line eLIFE.

Straightforward evidence that “HIV” is not sexually transmitted — in particular, not by heterosexual intercourse in Africa — is present in “Earlier menarche is associated with a higher prevalence of Herpes simplex type-2 (HSV-2) in young women in rural Malawi”, Glynn et al., eLife 2014;3:e01604, 28 January 2014.

The article’s main point is less than surprising: “girls with earlier menarche tend to have earlier sexual debut and school drop-out, so an association might be expected” with being more likely to contract sexually transmitted infections (STIs or STDs).
That expectation was confirmed by a close-to-linear relationship between age at menarche and prevalence of herpes (HSV-2) infection:


By contrast, there was no correlation at all between “HIV-positive” and age at menarche.

Furthermore, prevalence of “HIV” was much lower than that of HSV-2, contrary to yet another shibboleth of HIV/AIDS theory, namely, that infections by an STD like HSV-2 makes “HIV-positive” more likely:


In an earlier article (Glynn et al., “Assessing the validity of sexual behaviour reports in a whole population survey in rural Malawi”, PLoSONE, 27 July 2011) the ratio of “HIV-positive” to HSV-2 infection had been reported as 4/31 for females and 2/52 for males, again confirming that “HIV-positive” is much less prevalent than HSV-2.

Not, of course, that this further evidence that “HIV” isn’t an STD will make any difference, more-than-ample evidence has been around for many years.

Posted in clinical trials, HIV risk groups, HIV skepticism, HIV varies with age, sexual transmission | Tagged: , , | Leave a Comment »

Updates to The Case against HIV

Posted by Henry Bauer on 2014/03/25

The following additions have been made to The Case against HIV Co-factors in addition to HIV required to bring on AIDS have been postulated on a number of occasions: mycoplasma (refs. 26-31); HTLVs (p. 248 in ref. 257); cell surface protein CD26 (Callebaut et al., “T-cell activation antigen CD26, as a cofactor for entry of HIV into CD4+ cells”, Science, 262 [1993] 2045; Jon Cohen, “HIV ‘cofactor’ comes in for heavy fire”, Science, 262 [1993] 1971); the protein fusin (Jon Cohen, “Likely HIV cofactor found, Science, 272 [1996] 809-10) Malnutrition is widespread in Africa and is a known cause of lack of resistance to infection. It can be responsible for any infection incurred within 1 month of the end of food deprivation.
”Acquired Immunodeficiency Syndrome (AIDS) — WHO/CDC case definition for AIDS”, Weekly Epidemiological Record, 7 March 1986, 69-73 Fever is very non-specific. “HIV-positive” is often associated with fever.
Celia Farber, “Positively Flawed: Welcome to the Machine”, Impression, June 1999;
AND ALSO ADD THIS REFERENCE TO FOR PREGNANCY In Africa, more than two thirds of couples are serodiscordant: one is “HIV-positive”, the other is not
UNAIDS 2008 Report on the Global AIDS Epidemic, p. 118

3.3.18 add ref 465 In 1995, Peter Godwin, head of the Regional HIV Project, United Nations Development Program: “by the year 1997 the annual number of new HIV infections in Asia will exceed those in Africa, and its share of worldwide cumulative infections will increase to nearly 25% by the year 2000”
J. C., “Thailand points the way”, Science, 270 (1995) 30
but by 2007, Asia had only 5 out of a global 33 million “HIV-positives” while sub-Saharan Africa had 19 million
UNAIDS 2008 Report on the Global AIDS Epidemic Trojan Horse inhibitors
National Institute of Allergy and Infectious Diseases, “Trojan Horse Virus controls HIV infection”, 4 September 1997;
Tom Nurre, “Radical research”, Angelo State University Magazine, Summer 2011, 16-9
Buckley et al., “HIV protease-mediated activation of sterically capped proteasome inhibitors and substrates”, Journal of the American Chemical Society, 133 (2011) 698-700 Killing “HIV” by shaking it with tuned laser
Orli Van Mourik, “Good vibrations vs. bad viruses”, Discover, June 2008, p. 16

4.2.6 Add Cavrois, Neidleman & Greene, “The Achilles Heel of the Trojan Horse Model of HIV-1 trans-infection”, PLoS Pathogen, 4(#6, 2008): e1000051 Cardiovascular disease
refs 34, 120-5, 131-2 in Pinzone et al., Vitamin D deficiency in HIV infection: an underestimated and undertreated epidemic, European Review for Medical and Pharmacological Sciences, 17 (2013) 1218-1232 Bone disease, osteopenia, osteoporosis
refs. 35-43, 103 in Pinzone et al., Vitamin D deficiency in HIV infection: an underestimated and undertreated epidemic, European Review for Medical and Pharmacological Sciences, 17 (2013) 1218-1232

4.7.6 add another reference: UNAIDS 2008 Report on the Global AIDS Epidemic, p. 89 Vitamin D deficiency
Refs 74, 76, 77, 84, 89, 93-100, 102 in Pinzone et al., Vitamin D deficiency in HIV infection: an underestimated and undertreated epidemic, European Review for Medical and Pharmacological Sciences, 17 (2013) 1218-1232
Add Refs. 35, 38, 39, 41, 104-6, 111-6 in Pinzone et al., Vitamin D deficiency in HIV infection: an underestimated and undertreated epidemic, European Review for Medical and Pharmacological Sciences, 17 (2013) 1218-1232

6.1.3 Add “and from other experimental treatments”. Chemotherapy to kill the immune system to avoid rejection of transplanted baboon bone-marrow cells because baboons couldn’t be infected with “HIV”. (Rick Weiss, “Doctor hopes to repeat baboon cell transplant”, Washington Post, 11 February 1996, p. A9) The first attempt involved “an unidentified 56-year-old man who was dying of AIDS. . . . The experiment was not a success. The baboon cells failed to grow, and the man died two months later. But Dr. Ricordi said his team had been encouraged because the man did not suffer any adverse reactions from the transplant” [emphasis added].
Lawrence K. Altman, “The Doctor’s World; Baboon cells might repair AIDS-ravaged immune systems”, New York Times, 19 July 1994

6.4 Hegemony of HIV/AIDS theory and practices means that some central questions cannot be answered
6.4.1 When “HIV-positive” individuals become ill, all too often the real cause of illness is not looked for
6.4.2 When “HIV-positive” individuals using antiretroviral drugs die, no autopsy inquires into whether death may have been owing to the drugs
”42-yr-old Glen Elder, dead of heart attack at 42”, Chronicle of Higher Education, 12 June 2009, A33
” Jeff Getty, 49, AIDS activist who received baboon cells, is dead”, New York Times, 16 October 2006;
AND ADD THIS LAST REF. TO That many hemophiliacs were infected by tainted blood products
”Two thirds of our hemophiliacs have received infected blood”
Jonathan Kellerman, Devil’s Waltz, Bantam 1993, p. 336 The mistaken belief that “HIV” is transmitted sexually (see Section 3.3 for disproof) is incessantly repeated, for example that prostitutes contract and spread “HIV”
Colin Thubron, “Believers’ bazaar”, review of In Search of the Sacred in Modern India by William Dalrymple, New York Times Book Review, 18 July 2010, p. 19
Gurav & Blanchard, “Disease, Death and Dhandha: Gharwali’s Perspectives on the Impact of AIDS on Devadasi System and the Sex Work in South India”, World Journal of AIDS, 3 (2013) 26-32

8.3 Add “; see also Sections 7.1, 7.2, 7.4, 7.7”

Posted in HIV does not cause AIDS, HIV skepticism | Tagged: | Leave a Comment »

The Drug-AIDS hypothesis — a footnote

Posted by Henry Bauer on 2014/03/17

Peter Duesberg is usually credited with the “Drug-AIDS hypothesis”, namely, that AIDS was not and is not caused by HIV but is caused primarily by “recreational” and antiretroviral drugs [1].

Gordon Stewart had observed in the 1960s that drug addicts showed the same symptoms as were ascribed a couple of decades later to “AIDS” [2].

Nowadays, it is presumed that drug abusers contract “HIV” by sharing infected needles, as though the contents of the needles were harmless. My footnote refers to a review [3] of Breaking Night by Liz Murray: Murray’s parents were lifelong drug abusers who “usually burned through their monthly welfare check within a week, spending the money on cocaine”. Murray’s mother “died of AIDS at 42”. No “HIV” was needed, rather obviously.

So nowadays one cannot die just from drug abuse, it must be “AIDS”.


[1] Duesberg, Koehnlein, & Rasnick, The chemical bases of the various AIDS epidemics: recreational drugs, anti-viral chemotherapy and malnutrition, Journal of Bioscience, 28 (2003) 383-412
[2] Neville Hodgkinson, AIDS: The failure of contemporary science, Fourth Estate, 1996, p.103
[3] Tara McKelvey, Unsentimental education — review of Breaking Night, New York Times Book Review, 12 September 2012, p. 16

Posted in HIV absurdities, HIV does not cause AIDS, HIV risk groups, HIV skepticism, uncritical media | Tagged: , | Leave a Comment »

HIV/AIDS as pseudo-science

Posted by Henry Bauer on 2014/03/15

A vast literature in science studies, philosophy of science, history of science, sociology of science, and a variety of popular media deals with “pseudo-science”; sometimes enumerating instances of pseudo-science, sometimes focusing on a single example (creationism, or UFOs, etc, etc.) and sometimes grappling with an issue over which no consensus has been achieved despite a century or more of discussion: How to define pseudo-science? How to distinguish pseudo-science from real science?

The simplest identification of pseudo-science, with which almost no one would disagree (“there’s always one ….”), is something totally incompetent but pretending to be properly scientific. Examples can rather easily be found in “HIV/AIDS research”, for instance that one person has staved off the disease since 1978, which is several years before AIDS had even been identified and half-a-dozen years before “HIV” had been suggested as its cause (Mainstream HIV PSEUDO-science).

A commonly advanced supposed criterion for pseudo-science, directed often particularly at parapsychology, is an inability to reproduce results exactly. By that standard, HIV/AIDS “science” again qualifies as pseudo-science (HIV/AIDS and parapsychology: science or pseudo-science?)  — as of course does all of sociology and almost all of psychology.

My own empirically-based suggestion for when mainstream sources treat something as pseudo-science is if it differs from mainstream science in all the three aspects of established method, currently accepted fact, and standard theory. Once again, HIV/AIDS “science” qualified as pseudo-science (Defining pseudo-science: Three strikes against HIV/AIDS theory)  — that is, before it came to be accepted as unassailable dogma, which it was for reasons of politics and social factors, not on scientific grounds.

Yet another common criterion for supposedly characterizing pseudo-science, again often directed at parapsychology, is that claimed phenomena or events are not accompanied by the offering of any reasonable mechanism that could possibly cause those events or phenomena; see, for instance, Erich Goode, “Paranormalism and pseudoscience as deviance”, chapter 8, pp. 145-164 in Philosophy of Pseudoscience, University of Chicago Press, 2013.
Once more, HIV/AIDS qualifies as pseudo-science under that criterion. For three decades suggestions have been put forward as to how the purported retrovirus “HIV” could act to destroy the immune system. First it was found that some sort of direct action on the supposedly most involved immune-system cells (CD4 T-cells) was not a viable explanation. Subsequently a variety of indirect or “bystander” mechanisms have been suggested, all of which are notably non-specific, mere names like “chronic inflammation” unaccompanied by specific mechanisms (The Pathogenesis of AIDS).

By every measure that has been suggested for distinguishing science from pseudo-science, HIV/AIDS qualifies as pseudo-science.


P.S., after a useful comment sent to me privately:

As always, my intent here has been to undermine the pretensions of HIV/AIDS theory.
However, I also have quarrels with the very use of the term “pseudo-science”, because of the lack of agreed substantive meeting for the term; it is in practice not an objective label but a term of abuse. I’ve written a great deal about improper labeling of a number of matters as pseudo-science, and the fact that what was once called pseudo-science has sometimes become accepted science, and vice versa (Science or Pseudoscience: Magnetic Healing, Psychic Phenomena, and Other Heterodoxies, University of Illinois Press, 2001).

That parapsychology in particular has been called pseudo-science under a couple of the above-mentioned criteria should not be taken to mean that I think it is pseudo-science. However, I do think HIV/AIDS theory is dead wrong and deserves all sorts of pejorative labels.

Posted in HIV absurdities, HIV does not cause AIDS, HIV skepticism, uncritical media | Tagged: | 11 Comments »

Race, HIV, media pundits

Posted by Henry Bauer on 2014/03/09

People carrying black-African genes test “HIV-positive” at far greater rates than do people without that genetic ancestry. HIV/AIDS theory “explains” that by postulating greater rates of careless “not-safe-sex” promiscuity and infected-needle-sharing drug injection. Thereby HIV/AIDS theory postulates significant genetic determination of behavior, which in other contexts is dismissed as pseudo-science.

Moreover, actual observations and studies have repeatedly shown that the facts vitiate that proposed “explanation”: Africans and African-Americans indulge in risky behavior at lower rates than do white Americans (pp. 77-9 in The Origin, Persistence and Failings of HIV/AIDS Theory).
The conclusion is inescapable: HIV/AIDS theory is radically wrong about how “HIV-positive” is transmitted.

But that inescapable conclusion continues to escape mainstream practitioners and researchers and such media pundits as Donald G. McNeil Jr. of the New York Times (Poor Black and Hispanic men are the face of H.I.V.):

“The AIDS epidemic in America is rapidly becoming concentrated among poor, young black and Hispanic men who have sex with men”
NO. There’s nothing recent or rapid about it. The racial disparities have always been there (Chapters 5 & 6 in The Origin, Persistence and Failings of HIV/AIDS Theory).
Furthermore, it is black WOMEN who are most affected compared to others, 20 times more likely to be “HIV-positive” than white women, whereas for males the ratio is (“only”) 7.

“Nationally, 25 percent of new infections are in black and Hispanic men, and in New York City it is 45 percent”
Yes, of course, because it’s blackness that contributes overwhelmingly to testing “HIV-positive”. Hispanics in New York are primarily of black Caribbean-African stock, whereas West-Coast Hispanics are largely non-black, of Latin-American stock. Therefore national-average rates of “HIV-positive” among Hispanics are lower than East-Coast Hispanic rates of “HIV-positive” (pp. 57-8, 71-2 in The Origin, Persistence and Failings of HIV/AIDS Theory).

“Nationally, when only men under 25 infected through gay sex are counted, 80 percent are black or Hispanic — even though they engage in less high-risk behavior than their white peers” [emphasis added]; “a male-male sex act for a young black American is eight times as likely to end in H.I.V. infection as it is for his white peers. That is true even though, on average, black youths in the study took fewer risks than their white peers: they had fewer partners, engaged in fewer acts of sex while drunk or high, and used condoms more often”.
So McNeil is even aware of this conundrum which falsifies the central axiom of HIV/AIDS theory, namely, that HIV is transmitted as a result of risky behavior. Yet he does not follow this statement of fact with any explanation of this paradox which contradicts and falsifies mainstream views.
Instead, McNeil passes on without comment the usual meaningless weasel-words about some unspecified “intervention”:
“Critics say little is being done to save this group, and none of it with any great urgency. ‘There wasn’t even an ad campaign aimed at young black men until last year — what’s that about?’. Phill Wilson, president of the Black AIDS Institute in Los Angeles, said there were ‘no models out there right now for reaching these men’”.
What conceivable use could any models be, when it’s acknowledged that these supposedly at-high-risk people already practice less risky behavior than the no-high-risk white folk?
Still, of course there’s no harm in asking for more money even in absence of any clue what to do with it:
“With more resources, we could make bigger strides”.

What the mainstream says about the high rates of black “HIV-positives” is pitifully, woefully inadequate; it misses the whole point. It suggests that although their behavior is less risky, black folk have “other risk factors. Lacking health insurance, they were less likely to have seen doctors regularly and more likely to have syphilis, which creates a path for H.I.V.”
But it’s yet another counterfactual canard that syphilis and other STDs make it more likely that someone will “contract” “HIV”, i.e. become “HIV-positive”: there is simply no correlation between incidence of STDs and of “HIV” (pp. 31-5, 109 in The Origin, Persistence and Failings of HIV/AIDS Theory).
As to insurance, what is the evidence that having health insurance makes for lower rates of being or becoming “HIV-positive”? This is simply hand-waving bullshit* emitted because no sensible explanation can be offered.
As to seeing doctors regularly, what is the evidence that seeing doctors regularly makes for lower rates of being or becoming “HIV-positive”? Quite the opposite, in fact: The largely white gay men who first contracted “AIDS” had mostly been seeing doctors very often because of their constant need for treatment after suffering all sorts of illnesses. Dr. Joseph Sonnabend, with a practice of largely gay clients in New York in the 1970s, had in fact warned his regular customers that if they did not change their lifestyle something drastic and awful would befall them.

And then, “Other risk factors include depression and fatalism” — What, pray, is the mechanism by which those conditions produce “HIV-positive”? Among people who are acknowledged to behave less riskily than those who are not at high risk of becoming “HIV-positive”?

Another popular non-explanation is that blacks become “HIV-positive” more often because “HIV-positive” is so much more common in the black community: It’s more common because it’s more common.

I cannot imagine a higher degree of hypocrisy, intellectual vapidity, sheer unwillingness to draw obvious conclusions from undisputed facts, than is demonstrated without fail and without end by mainstream researchers, doctors, and pundits when confronted with the plain fact that blackness makes for being “HIV-positive”.

Not that this perverse behavior is much different from behaving as though testing “HIV-positive” proved infection by “HIV” when standard authorities have long stated quite forthrightly that there is no gold standard “HIV” test, no test capable of demonstrating actual infection by “HIV”, and that the rates of false positives are inevitably high (Stanley H. Weiss & Elliot P. Cowan, “Laboratory detection of human retroviral infection”, chapter 8 in Gary P. Wormser (ed.), AIDS and Other Manifestations of HIV Infection, 2004 (4th ed.).

No technical expertise is needed to recognize the sheer unadulterated nonsense of talking about “risk factors” when the known end-result is less risky behavior. How can any number of purported risk factors be alleged to heighten risk when the facts show that the risk is lower of the only behavior that supposedly transmits “HIV”?

* Words uttered without regard to their truth — Harry Frankfurt, On Bullshit, Princeton University Press, 2005.

Posted in experts, HIV absurdities, HIV and race, HIV risk groups, HIV skepticism, HIV tests, HIV transmission, HIV/AIDS numbers, prejudice, sexual transmission, uncritical media | Tagged: , , | 4 Comments »


Get every new post delivered to your Inbox.

Join 109 other followers